Toe-Brachial Index (TBI) in Non-Compressible Arteries (Diabetes, CKD): Methodology and Thresholds — МЕДТРЕЙН Asia
Angiology

Toe-Brachial Index (TBI) in Non-Compressible Arteries (Diabetes, CKD): Methodology and Thresholds

Briefly. In patients with diabetes and CKD (medial calcinosis), non-compressible arteries cause the ABI to appear falsely normal or high (>1.40) and do not rule out PAD. In these cases, the Toe-Brachial Index (TBI) is performed — the ratio of the pressure on the first toe to the highest brachial pressure; a threshold value of <0.70 is considered pathological.

Non-Compressibility Phenomenon and TBI Rationale

In patients with diabetes, chronic kidney disease (CKD), and the elderly, medial calcinosis — calcification of the medial layer of the tibial arteries — develops. The wall loses compressibility, the cuff does not occlude the vessel, and the ABI appears falsely normal or even high (>1.40) even with confirmed severe lesions. Since toe arteries are almost unaffected by medial calcinosis, the Toe-Brachial Index (TBI) retains diagnostic value where the ABI is uninformative.

Indications for TBI

TBI is performed when the ABI is >1.40 (a sign of non-compressibility), as well as generally in patients with diabetes where a normal or high ABI does not rule out peripheral artery disease (PAD).

Methodology and Interpretation

TBI is the ratio of the pressure on the first toe to the highest brachial pressure. A threshold value of <0.70 is considered pathological and allows for the diagnosis of PAD in situations where the ABI is uninformative.

IndicatorValueInterpretation
ABI<0.90Peripheral Artery Disease
ABI>1.40Non-Compressibility (TBI indicated)
TBI<0.70Pathology (PAD)

Diagnostic Value in Diabetics

In patients with diabetes, TBI is significantly more sensitive than ABI: compared to CT angiography with a formally “normal” ABI, the sensitivity of TBI is about 80% versus about 35% for ABI.

Place in Ischemia Assessment System

ABI and TBI are fundamental to the quantitative assessment of ischemia. The thresholds of ABI/TBI as a measure of the ischemic component are the basis for all classifications of ischemia severity: both the classic clinical stages (Fontaine, Pokrovsky) and the modern WIfI system.

Frequently asked questions

At what ABI is TBI indicated?

When the ABI is >1.40 (a sign of non-compressibility), as well as in patients with diabetes where a normal or high ABI does not rule out PAD.

What TBI threshold is considered pathological?

A TBI <0.70 is considered pathological and allows for the diagnosis of PAD where the ABI is uninformative.

How is TBI calculated?

TBI is the ratio of the pressure on the first toe to the highest brachial pressure.

Why is TBI more informative than ABI in diabetics?

Toe arteries are almost unaffected by medial calcinosis. The sensitivity of TBI with a “normal” ABI is ~80% versus ~35% for ABI (compared to CT angiography).

In which patients is the ABI falsely normal or high?

In patients with diabetes, CKD, and the elderly, due to medial calcinosis of the tibial arteries, the wall loses compressibility, and the ABI appears falsely normal or high (>1.40).

The material is intended for specialists and does not replace clinical judgment. Threshold values are periodically reviewed — refer to the current edition of the applicable consensus.
Sources: Blagodir B.V. Ultrasound Examination of Vessels: A Modern Practical Guide, 2026; ESVS 2024 Clinical Practice Guidelines on the Management of Asymptomatic Lower Limb Peripheral Arterial Disease and Intermittent Claudication, 2024; IWGDF/ESVS/SVS guidelines on peripheral artery disease in patients with diabetes mellitus and a foot ulcer, 2023; 2024 ACC/AHA/AACVPR/APMA/ABC/SCAI/SVM/SVN/SVS/SIR/VESS Guideline for the Management of Lower Extremity Peripheral Artery Disease, 2024.
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